EU PHARMACEUTICALS TEST ENANTHATE 250
EU PHARMACEUTICALS TEST ENANTHATE 250(250MG/ML TESTOSTERONE ENANTHATE=10ML)
Active-Life: 8 days
Drug Class: Androgenic/Anabolic Steroid (For injection)
Average Reported Dosage: Men 200-1000mg weekly.
Water Retention: Yes, high due to estrogen conversion
High Blood Pressure: Yes, normally due to high water /electrolyte retention
Liver Toxic: Low in listed dosages
Aromatization: Yes, high
DHT Conversion: Yes, high
Decreases HPTA Function: Yes, high
Testosterone was generally toted as the big daddy of injectable steroids. No other steroid was consistently reported to bring such high returns as quickly in weight gain and strength increases. Due to its high anabolic/high androgenic effects, many athletes used this drug in an off-season mass cycle. Water retention during administration of ENANTHATE was not reportedly as high as that realized during the use of OMNADREN. but darn close. Like all testosterone esters, Enanthate aromatized easily and has a high conversion rate to DHT. Those with prostate problems or who were sensitive to gyno and female pattern fat deposits, readily agreed that they should have either left it alone or taken steps to suppress estrogenic activity due to aromatization.
Drugs such as PROVIRON and NOVLADEX were often utilized for this reason. DHT
conversion enzyme blockers such as Proscar were commonly co-administered with testosterones for the former reason.
Testosterone enanthate notably suppressed HPTA function severely. HCG/Clomid were considered almost a must to stimulate normal endogenous (natural) testosterone production within a positive period of time at post use. My personal experience has been that if a cycle containing testosterone enanthate lasted longer than 6 weeks, HCG and usually Clomid were introduced for 10 days beginning at the end of week #4. (5000 i.u. of HCG 3 times in 10 days usually normalized sperm and endogenous testosterone production to a respectable extent) Without the use of HPTA stimulating compounds normalization did occur, only at a much slower rate. For this reason, gains made during "enanthate only" administrations were not well maintained after use was discontinued, and much was lost needlessly by most regardless. Perhaps this was why so many uninformed individuals stayed on the stuff almost year round. (There are several solutions and protocols that prevented excessive post-cycle lean mass tissue loss for the more informed athletes)
Males injected 200-1000mg weekly. Some did use much higher dosages of course. Due to a plasma half-life of 4-5 days, injections were normally administered biweekly. Most novice steroid should not use testosterone. Not only was considered unnecessary, it would have been foolish to diminish possible later gains when more gentle AAS were no longer providing results at reasonable dosages. Most users made excellent progress with a total weekly dosage of 200-600mg. Post-cycle use of an anti-catabolic drug was a constant agreed upon factor since it helped to maintain gains. (See Clenbuterol)
The negative side effects reported were mostly water retention and strong androgenic effects. These included gyno, accelerated hair growth, receding hair-lines, aggressiveness, higher blood pressure, acne, and increased fat deposits (due to aromatization). Since testosterones are metabolized by the liver fairly easily, alarming elevated liver enzymes occurred in very high dosages only. usually.
Androgens are derivatives of cyclopentano-perhydrophenanthrene. Endogenous androgens are C-19 steroids with a side chain at C-17, and with two angular methyl groups. Testosterone is the primary endogenous androgen.
In their active form, all drugs in the class have a 17-beta-hydroxy group. Esterification of the 17-beta-hydroxy group produces compounds (testosterone enanthate and testosterone propionate) which have a longer duration of action and are hydrolyzed in vivo to free testosterone.
Androgens are steroids that develop and maintain primary and secondary male sex characteristics.
Endogenous androgens are responsible for the normal growth and development of the male sex organs and for maintenance of secondary sex characteristics. These effects include the growth and maturation of prostate, seminal vesicles, penis, and scrotum; the development of male hair distribution, such as beard, pubic, chest and axillary hair; laryngeal enlargement, vocal chord thickening, alterations in body musculature, and fat distribution. Drugs in this class also cause retention of nitrogen, sodium, potassium, phosphorous, and decreased urinary excretion of calcium. Androgens have been reported to increase protein anabolism and decrease protein catabolism. Nitrogen balance is improved only when there is sufficient intake of calories and protein.
Androgens are responsible for the growth spurt of adolescence and for the eventual termination of linear growth which is brought about by fusion of the epiphyseal growth centers. In children, exogenous androgens accelerate linear growth rates, but may cause a disproportionate advancement in bone maturation. Use over long periods may result in fusion of the epiphyseal growth center and termination of growth process.
Androgens have been reported to stimulate the production of red blood cells by enhancing the production of erythropoietic stimulating factor.
During exogenous administration of androgens, endogenous testosterone release is inhibited through feedback inhibition of pituitary luteinizing hormone (LH). At large doses of exogenous androgens, spermatogenesis may also be suppressed through feedback inhibition of pituitary follicle stimulating hormone (FSH).